software corel draw Search Results


corel draw 2018 version 20.1.0.708 software  (corel corporation)
90
corel corporation corel draw 2018 version 20.1.0.708 software
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Corel Draw 2018 Version 20.1.0.708 Software, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/corel draw 2018 version 20.1.0.708 software/product/corel corporation
Average 90 stars, based on 1 article reviews
corel draw 2018 version 20.1.0.708 software - by Bioz Stars, 2026-04
90/100 stars
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corel draw graphic suite ×5 software  (corel corporation)
90
corel corporation corel draw graphic suite ×5 software
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Corel Draw Graphic Suite ×5 Software, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/corel draw graphic suite ×5 software/product/corel corporation
Average 90 stars, based on 1 article reviews
corel draw graphic suite ×5 software - by Bioz Stars, 2026-04
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corel draw computer software version x8  (corel corporation)
90
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Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Corel Draw Computer Software Version X8, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/corel draw computer software version x8/product/corel corporation
Average 90 stars, based on 1 article reviews
corel draw computer software version x8 - by Bioz Stars, 2026-04
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coral draw x4 software (ver 14.0.0.701  (corel corporation)
90
corel corporation coral draw x4 software (ver 14.0.0.701
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Coral Draw X4 Software (Ver 14.0.0.701, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/coral draw x4 software (ver 14.0.0.701/product/corel corporation
Average 90 stars, based on 1 article reviews
coral draw x4 software (ver 14.0.0.701 - by Bioz Stars, 2026-04
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computer-aided design software coreldraw  (corel corporation)
90
corel corporation computer-aided design software coreldraw
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Computer Aided Design Software Coreldraw, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/computer-aided design software coreldraw/product/corel corporation
Average 90 stars, based on 1 article reviews
computer-aided design software coreldraw - by Bioz Stars, 2026-04
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imaging software package corel draw v10.0  (corel corporation)
90
corel corporation imaging software package corel draw v10.0
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Imaging Software Package Corel Draw V10.0, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/imaging software package corel draw v10.0/product/corel corporation
Average 90 stars, based on 1 article reviews
imaging software package corel draw v10.0 - by Bioz Stars, 2026-04
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corel draw software coral draw graphics suite x3  (corel corporation)
90
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Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Corel Draw Software Coral Draw Graphics Suite X3, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/corel draw software coral draw graphics suite x3/product/corel corporation
Average 90 stars, based on 1 article reviews
corel draw software coral draw graphics suite x3 - by Bioz Stars, 2026-04
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dimension tools of the software corel draw 2017  (corel corporation)
90
corel corporation dimension tools of the software corel draw 2017
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Dimension Tools Of The Software Corel Draw 2017, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/dimension tools of the software corel draw 2017/product/corel corporation
Average 90 stars, based on 1 article reviews
dimension tools of the software corel draw 2017 - by Bioz Stars, 2026-04
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computer software corel draw graphics suits x5  (corel corporation)
90
corel corporation computer software corel draw graphics suits x5
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Computer Software Corel Draw Graphics Suits X5, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/computer software corel draw graphics suits x5/product/corel corporation
Average 90 stars, based on 1 article reviews
computer software corel draw graphics suits x5 - by Bioz Stars, 2026-04
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dimensioning tool of the corel draw software package  (corel corporation)
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corel corporation dimensioning tool of the corel draw software package
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Dimensioning Tool Of The Corel Draw Software Package, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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corel draw software package v.12.0  (corel corporation)
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corel corporation corel draw software package v.12.0
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Corel Draw Software Package V.12.0, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/corel draw software package v.12.0/product/corel corporation
Average 90 stars, based on 1 article reviews
corel draw software package v.12.0 - by Bioz Stars, 2026-04
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computer software corel draw® graphics suite x3  (corel corporation)
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corel corporation computer software corel draw® graphics suite x3
Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw <t>2018</t> version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.
Computer Software Corel Draw® Graphics Suite X3, supplied by corel corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/computer software corel draw® graphics suite x3/product/corel corporation
Average 90 stars, based on 1 article reviews
computer software corel draw® graphics suite x3 - by Bioz Stars, 2026-04
90/100 stars
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Image Search Results


Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw 2018 version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.

Journal: Scientific Reports

Article Title: Differential epigenetic regulation between the alternative promoters, PRDM1α and PRDM1β , of the tumour suppressor gene PRDM1 in human multiple myeloma cells

doi: 10.1038/s41598-020-72946-z

Figure Lengend Snippet: Schematic representation of the regulation of human PRDM1 promoters in multiple myeloma. a Factors such as SP1, SP3 and EGR1 act on the PRDM1α promoter, inducing its transcription , the opposite effect as that of PAX5 . On the PRDM1β promoter, TP53 (as an activator) , and FOX factors (as inhibitors) can regulate PRDM1β transcription. Regarding epigenetic regulation, methylation seems to play a decisive role in PRDM1β expression but not in PRDM1α expression. In the case of histone modifications, H3K4me1 and H3K9me2 marks seem to be linked to the repression of the PRDM1β promoter. Activators and inhibitors are represented above and under the DNA helix, respectively. Filled and open lollipops represent the methylation and demethylation status of the promoter, respectively. This figure was created using Corel Draw 2018 version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate. Inhibiting PRDM1β expression using drugs targeting epigenetic elements at its promoter may restore the balance to maintain normal PC cell fate.

Article Snippet: This figure was created using Corel Draw 2018 version 20.1.0.708 software (Corel Corporation, Ottawa, Canada). b Schematic representation showing how PRDM1β expression tilts the balance in favour of proliferation due to a diminished apoptosis rate.

Techniques: Methylation, Expressing, Software